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Excess circulating endotoxin then activates Kupffer cells through interaction with Toll-like Receptor 4 (TLR4) to set off the inflammatory cascade responsible for the development of alcoholic liver disease

An efficacious treatment for AD has not been forthcoming in part due to the large numbers of potentially important biomarkers, the difficulty in determining their diagnostic reliability and validity, and the multifactorial characteristics of the disease process

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