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We hypothesized that smoke will activate tracheal inflammatory, apoptotic, proliferative, and stress-induced pathways

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C-EBP mediates in CSE/IL-17A-induced epithelialmesenchymal transition of bronchial epithelial cells When C-EBP was interfered, the E-cadherin protein expression in CSE group (Fig

Table 2 shows the results of the adjusted ordinal logistic regression models