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Genomic analysis revealed oxidative stress-associated mutational patterns, suggesting that continuous exposure imprints both functional and genetic adaptations

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28 proposed in a meta-analysis that the odds for AP among ex-smokers remained significantly higher than among non-smokers (HR=1.63

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The number of genes differentially expressed between smokers and nonsmokers differed depending on the tissue, with most differences occurring in the lungs, pancreas, thyroid, and the cells lining the esophagus